Dysmenorrhea after uterine artery embolization

Dysmenorrhea after uterine artery embolization may be related to postoperative tissue ischemia, inflammatory response or intrauterine adhesions. If the symptoms continue to worsen or are difficult to relieve, it is recommended to seek medical attention in time to evaluate the cause and treatment plan.

The occurrence of postoperative dysmenorrhea is closely related to changes in blood circulation in the body. Uterine artery embolization blocks the arteries of uterine fibroids or abnormal blood supply areas, causing the corresponding tissues to degenerate, necrotize, and shrink. This process may cause ischemic pain in the surrounding tissues, especially when blood flow changes during menstruation. Local inflammatory reactions after surgery may also stimulate the contraction of the uterine wall and aggravate dysmenorrhea. At the same time, some patients may experience dysmenorrhea due to postoperative endometrial adhesions or scar tissue formation. Whether it is short-term tissue adaptive changes or potential surgical complications, it is recommended to adjust the response measures in a timely manner according to the degree and fluctuation characteristics of dysmenorrhea, combined with postoperative evaluation.

The occurrence of postoperative dysmenorrhea is closely related to changes in blood circulation in the body. Uterine artery embolization blocks the arteries of uterine fibroids or abnormal blood supply areas, causing the corresponding tissues to degenerate, necrotize, and shrink. This process may cause ischemic pain in the surrounding tissues, especially when blood flow changes during menstruation. Local inflammatory reactions after surgery may also stimulate the contraction of the uterine wall and aggravate dysmenorrhea. At the same time, some patients may experience dysmenorrhea due to postoperative endometrial adhesions or scar tissue formation. Whether it is short-term tissue adaptive changes or potential surgical complications, it is recommended to adjust the response measures in a timely manner according to the degree and fluctuation characteristics of dysmenorrhea, combined with postoperative evaluation.

There are many ways to relieve postoperative dysmenorrhea: symptomatic medications such as ibuprofen or acetaminophen can effectively relieve discomfort caused by inflammatory stimulation; hormonal drugs that regulate menstruation (such as Zoladex or levonorgestrel intrauterine device) can reduce menstrual blood volume and uterine contractions; physical therapy such as warm compresses to solve pain caused by ischemia can also help. Adjusting your lifestyle, including regular work and rest, light aerobic exercise (such as yoga or brisk walking) and avoiding cold stimulation can also help reduce the frequency and intensity of dysmenorrhea. If symptoms do not improve for a long time, you should seek help from a professional doctor to check for potential intrauterine adhesions or other lesions, and choose interventional treatment, intrauterine surgery to separate adhesions or further regulatory treatment according to your personal situation.