Let's take a look at vulvar leukoplakia.

Did you know that vulvar leukoplakia is a stubborn disease ? The unbearable itching and itching can affect the patient's work mood , affect normal communication with others , and seriously affect the couple's life . 10% of patients may develop cancer , and it is difficult to effectively eradicate other parts of the body . Now let's take a closer look at vulvar leukoplakia .

Clinical pathological examination shows atypical cell proliferation, which belongs to the category of " vulvar itching " , " vulvar pain " and " vulvar pain " in traditional Chinese medicine . It is mostly caused by the invasion of damp heat in the liver meridian into the vulva , or blood deficiency and liver hyperactivity, liver and kidney yin deficiency, kidney yang deficiency and other essence and blood cannot nourish the vulva . Vulvar itching , sometimes there may be burning and pain. Vulvar leukoplakia is also called vulvar white lesions , which is a chronic female vulvar malnutrition disease . It includes primary vulvar atrophy, atrophic lichen sclerosis, vulvar leukoplakia, leukoplakia vaginitis, and vulvar dryness . Clinicians usually refer to vulvar diseases that become white and thick and atrophic to the skin and sticky touch as vulvar leukoplakia . Chinese medicine calls it vulvar moss, vulvar sore, vulvar erosion, and vulvar pain . This disease is a precancerous lesion .

Benign keratotic lesions in the vulva are called white keratosis . White keratosis that does not heal for a long time can further develop into vulvar leukoplakia . The cause is unclear , and it may be related to systemic factors , such as diabetes and endocrine disorders . The local environment of the vulva , such as moisture, heat and other physical stimulations , can induce vulvar leukoplakia . Histopathology shows proliferative lesions of the mucosal epithelium or epidermis . Under normal circumstances , the vulvar mucosa has no granular layer and stratum corneum . If white keratosis or vulvar leukoplakia occurs, the granular layer will be thinned . If it grows in the transitional area of ​​the skin and mucosa of the labia majora, There is obvious hyperkeratosis , thickening of the granular layer , and hypertrophy of the spinous layer . About 80% of the lesions are benign white keratosis . There is no acanthosis atypia in the spinous cell layer , and there are generally no dyskeratotic cells. Chronic inflammatory cell infiltration is present under the epidermis . Early stage changes can be seen in vulvar leukoplakia , that is, some spinous cells are amorphous , the nuclear size is inconsistent , the cell size is also inconsistent, and the spinous cells are arranged in disorder . There are dyskeratotic cells in the superficial layer of spinous cells , which are called malignant dyskeratotic cells . There are varying degrees of inflammatory cell infiltration in the dermis, and there is no pure degeneration of collagen fibers in the superficial layer of the dermis .

The above is the introduction of relevant information about vulvar leukoplakia . I hope it will be helpful for your understanding . If you have other questions about vulvar leukoplakia , please consult our experts online and they will answer your questions . Thank you .

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