Treatment measures for shock in patients with ectopic pregnancy

Treatment measures for shock in patients with ectopic pregnancy

Patients with ectopic pregnancy suffer from acute intra-abdominal bleeding, which causes a decrease in blood volume and severe abdominal pain. Mild cases often cause syncope, while severe cases may experience shock. The treatment measures for patients with ectopic pregnancy who experience shock are as follows:

Take a V-shaped lying position

Raise the head and feet of the patient with ectopic pregnancy by 15°-30° respectively. This position is beneficial for unobstructed airway and increased venous return to the lower limbs, which is beneficial for blood supply to the body's important organs.

Replenish blood volume

If a patient with ectopic pregnancy is in shock, two or more intravenous channels should be established immediately to replenish blood volume. The fluid should be dripped in quickly, about 100 drops per minute, and 500ml-1000ml of fluid should be infused within 1 hour. When vasospasm occurs and fluid is difficult to infuse, pressurized infusion can be used.

Empty your bladder and test your urine output

Insert a urinary catheter to empty the bladder to avoid damaging the bladder during surgery. Urine can also be observed to understand the blood perfusion of major organs in the body and whether shock has improved.

Oxygen

According to the degree of shock, patients with ectopic pregnancy should be given an appropriate flow of oxygen to improve tissue hypoxia and facilitate recovery from shock.

Keep warm

Patients with shock from ectopic pregnancy have cold limbs and should raise the room temperature or use a quilt to keep warm to maintain normal body temperature. When transfusing blood, pay attention to warming the blood to avoid a large amount of low-temperature blood entering the body to cause spasm of arterioles and capillaries, causing microcirculatory disorders and further aggravating anaerobic metabolism and acidosis during the body weight period. It can also avoid the slow entry of liquid due to vascular spasm and narrowing of the lumen, thus prolonging the time of anti-shock.

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